A genetic glitch in one inhibitory receptor (FcγRIIB) stops it from working properly, so the immune system can’t calm down — leading to attacks on the body’s own tissues.

This study talks generally about how certain immune receptors work in diseases like lupus, but it doesn’t mention the specific genetic change (T232) or how it breaks the immune system’s brakes, so we can’t say if the claim is right or wrong based on this paper.