After the plaque burst, the number of immune cells that calm down inflammation went up — like the body’s way of trying to fix the damage after the explosion.

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Against

Evidence from Studies

Supporting (0)

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No supporting evidence found

Contradicting (1)

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Th17 cells and IL-17 are involved in the disruption of vulnerable plaques triggered by short-term combination stimulation in apolipoprotein E-knockout mice

Cohort Study

Animal

2013 Jul

This study found that after plaque rupture, the immune cells that calm inflammation (Tregs) didn’t increase at all — contrary to what the claim says. Instead, it found more of the cells that make inflammation worse.

Source Study

Th17 cells and IL-17 are involved in the disruption of vulnerable plaques triggered by short-term combination stimulation in apolipoprotein E-knockout mice

Score: 17

DOI: 10.1038/cmi.2013.4

Similar Assertions

When mice with fatty arteries were stressed, the number of a specific type of immune cell called Th17 went up dramatically — but another type, Th1, stayed the same, suggesting Th17 might be special in causing artery damage.

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