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The Study

POLYMORPHISMS IN THE SERUM- AND GLUCOCORTICOID-INDUCIBLE KINASE 1 GENE ARE ASSOCIATED WITH BLOOD PRESSURE AND RENIN RESPONSE TO DIETARY SALT INTAKE

In simple terms

This study found that people with certain versions of a gene (SGK1) tended to have higher blood pressure when they ate a lot of salt, but not when they ate little salt. It doesn’t prove the gene causes the high blood pressure — just that they’re linked in this group of people.

44%

Analysis score

44/ 44

Maximum 44 for a cross-sectional study.

Where the score came from

Reporting0
Methodology53
Publication100
Statistical54
Study type (basis of the score)
Cross-Sectional Study
Level 4 - Case series
What’s the bottom line?

Some people have a genetic quirk that makes their blood pressure go up when they eat too much salt, because their kidneys don't properly signal the body to slow down salt retention.

Where does this study sit?

Reviews of RCTs (Meta-analyses)

Max 100

Randomized Trials

Max 90

Reviews of Cohort Studies

Max 85

Cohort Studies

Max 72

Reviews of Case-Control Studies

Max 63

Case-Control Studies

Max 58

Cross-Sectional & Case Series

Max 50

Expert Opinion

Max 5
StrongerWeaker
Cross-Sectional & Case Series
Level 4
44

44 / 100

Quality score

Snapshots of a population at a single point in time, or descriptions of small groups. Can identify correlations and prevalence, but cannot determine cause and effect.

Cannot establish causation

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Key takeaways

Summary

Based on the study abstract and findings.

  1. 1Yes — a 16.3 mmHg BP spike on salt is clinically meaningful and increases heart disease risk.
  2. 2People with two copies of the SGK1 gene version had blood pressure rise 16.3 mmHg on high salt, vs.
  3. 39.1 mmHg in others.
  4. 4They also had 2.18x higher odds of low-renin hypertension.

Score breakdown, methodology, conflicts of interest, evidence analysis & raw study data

Publication

Journal

Journal of human hypertension

Year

2012

Authors

A. Rao, Bei Sun, A. Saxena, P. Hopkins, X. Jeunemaître, N. Brown, G. Adler, Jonathan S. Williams

Open Access
52 citations
Analysis v5

Related Content

Claims (6)

Assertion

People of Caucasian descent with high blood pressure who have two copies of certain gene versions may be more likely to have a type of high blood pressure that doesn’t respond well to the body’s normal salt-control system, and this could mean their condition is driven by how their genes affect salt and hormone balance.

Correlational
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Assertion

People of Caucasian descent with a certain common gene version might see their blood pressure rise a little when they eat a lot of salt, but the change is so small that we can’t be sure it’s real yet — we’d need to study more people to find out for sure.

Correlational
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Assertion

People of Caucasian descent with high blood pressure who have two copies of certain gene versions tend to see their blood pressure rise more when they eat a lot of salt, and their body produces less of a hormone called renin when they eat little salt—this suggests their genes might make them more sensitive to how much salt they eat.

Mechanistic
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Assertion

For people with high blood pressure who are white, having certain versions of the SGK1 gene might make their blood pressure more sensitive to salt—but only if they eat a lot of salt. If they eat very little salt, this gene doesn’t seem to affect their blood pressure at all.

Mechanistic
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Assertion

In people with high blood pressure who are white, certain gene variations seem to make their bodies produce less of a hormone called renin when they eat less salt—but these same gene changes don’t affect another hormone called aldosterone. This suggests the genes are tweaking renin on their own, not through aldosterone.

Correlational
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Assertion

Some people’s genes make them more likely to get high blood pressure when they eat salty foods, while others don’t — it’s all in their DNA.

Mechanistic
Read analysis
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