The Claim
Known genetic variants associated with lactase persistence explain only a portion of the trait's prevalence across populations in western and southern Africa, southeastern Europe, the Middle East, and parts of central and southern Asia, indicating that additional undiscovered genetic variants contribute to lactase persistence in these regions.
What the research says
Supports is higher
Support is ahead, but a single strong opposing study can change this.
These are independent scores, not a percentage. Higher-grade studies count more, so a single strong opposing study can outweigh several weaker ones.
In certain populations across Africa, Europe, the Middle East, and Asia, known genetic variants do not fully account for why some people can digest milk as adults, suggesting other genetic variants remain to be discovered.
See the scientific wording
Known genetic variants associated with lactase persistence explain only a portion of the trait's prevalence across populations in western and southern Africa, southeastern Europe, the Middle East, and parts of central and southern Asia, indicating that additional undiscovered genetic variants likely contribute to lactase persistence in these regions.
In some people, changes in DNA far from the lactase gene turn on the gene again after infancy, allowing the body to keep digesting milk sugar into adulthood.
What the research says
1 studyStudy: A worldwide correlation of lactase persistence phenotype and genotypes
Scientists found that the genes we already know about can't fully explain why so many people in Africa, Europe, and Asia can drink milk as adults — so there must be other hidden genes causing this ability.
Score breakdown, mechanism chain, raw evidence, ideal studies needed & 1 supporting studies
Not medical advice. For informational purposes only. Always consult a qualified healthcare professional before making health decisions.