The Claim

In individuals with type 1 or type 2 diabetes undergoing medication withdrawal, hypohydration increases postprandial blood glucose levels compared to euhydration, and this increase is attributable to reduced glucosuria rather than direct hormonal effects of arginine vasopressin or cortisol.

Source: Hydration, Arginine Vasopressin, and Glucoregulatory Health in Humans: A Critical Perspective

What the research says

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How it works
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In plain English

When people with type 1 or type 2 diabetes stop taking their medication and become dehydrated, their blood sugar after meals rises higher than when they are properly hydrated, and this rise is caused by less glucose being excreted in urine, not by changes in arginine vasopressin or cortisol levels.

See the scientific wording

In individuals with type 1 or type 2 diabetes undergoing medication withdrawal, hypohydration increases postprandial blood glucose compared to euhydration, but this effect is likely due to reduced glucosuria rather than direct hormonal effects of AVP or cortisol.

Why this might work

When a person with diabetes stops taking medication and becomes dehydrated, their kidneys stop removing as much sugar from the blood into the urine. This causes more sugar to stay in the bloodstream after eating, raising blood glucose levels. Hormones like AVP or cortisol do not cause this rise because they do not increase glucose production under normal dehydration conditions.

Verified mechanismbased on 1 study

What the research says

1 study
  1. Study: Hydration, Arginine Vasopressin, and Glucoregulatory Health in Humans: A Critical Perspective

    When people without diabetes get dehydrated, their blood sugar doesn’t go up — even though their body makes more of a hormone called AVP. This suggests that in diabetics who stop their meds, higher blood sugar after drinking water is probably because their kidneys aren’t flushing out as much sugar in urine, not because of hormones.

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