The Claim

Sulforaphane reduces the expression and activity of cytochrome P450 3A4 in human primary hepatocytes by antagonizing the steroid and xenobiotic receptor (SXR/hPXR), which normally induces this enzyme in response to xenobiotics.

Source: The Dietary Isothiocyanate Sulforaphane Is an Antagonist of the Human Steroid and Xenobiotic Nuclear Receptor

What the research says

Supports is higher

Support is ahead, but a single strong opposing study can change this.

Supports
20score
Challenges
0score

These are independent scores, not a percentage. Higher-grade studies count more, so a single strong opposing study can outweigh several weaker ones.

How it works
1 study reviewed
In plain English

Sulforaphane decreases the levels and function of the liver enzyme cytochrome P450 3A4 by blocking the receptor that activates this enzyme when the body encounters foreign substances.

See the scientific wording

Sulforaphane reduces the expression and activity of cytochrome P450 3A4 in human primary hepatocytes by antagonizing the steroid and xenobiotic receptor (SXR/hPXR), which normally induces this enzyme in response to xenobiotics.

Why this might work

Sulforaphane binds to a liver receptor that normally turns on a drug-processing enzyme, preventing the receptor from activating the gene for that enzyme. Without activation, the enzyme is not made in large amounts, so the liver breaks down fewer drugs.

Verified mechanismbased on 1 study

What the research says

1 study
  1. Study: The Dietary Isothiocyanate Sulforaphane Is an Antagonist of the Human Steroid and Xenobiotic Nuclear Receptor

    Sulforaphane, a compound in broccoli, blocks a liver receptor that normally tells the body to make more of a drug-breaking enzyme. This means the enzyme makes less, so drugs might stay in the body longer.

Score breakdown, mechanism chain, raw evidence, ideal studies needed & 1 supporting studies

Fit Body Science verdict — we translate health claims into clear verdicts backed by peer-reviewed research.

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