The Claim

Both glutamatergic and GABAergic neurons in the medial amygdala project to the ventromedial hypothalamus and can independently increase blood glucose levels when activated, indicating that a dual excitatory-inhibitory neural circuit regulates stress-induced hyperglycemia.

Source: Amygdala–liver signalling orchestrates glycaemic responses to stress

What the research says

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Supports
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Challenges
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How it works
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In plain English

When you're stressed, two types of brain cells in a specific area send signals to another part of the brain that tells your body to raise your blood sugar — even though one type excites and the other inhibits, both can make your blood sugar go up.

See the scientific wording

Both glutamatergic and GABAergic neurons in the medial amygdala project to the ventromedial hypothalamus and can independently increase blood glucose when activated, indicating a dual excitatory-inhibitory circuit controls stress-induced hyperglycemia.

Score breakdown, mechanism chain, raw evidence, ideal studies needed

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