The Claim
In baboons, administration of thyroid hormone increases total beta-adrenergic receptor density by more than 100% (from 15.7±3.2 to 37.4±1.2 fmol/mg) and preferentially upregulates the beta-2 subtype (from 5.9±1.5 to 20.6±1.2 fmol/mg) over the beta-1 subtype (from 9.7±1.7 to 16.8±0.1 fmol/mg).
What the research says
Supports is higher
Support is ahead, but a single strong opposing study can change this.
These are independent scores, not a percentage. Higher-grade studies count more, so a single strong opposing study can outweigh several weaker ones.
When baboons are given thyroid hormone, their bodies develop many more of a certain type of receptor that helps respond to stress and energy signals—especially the beta-2 kind—making them much more sensitive to these signals.
See the scientific wording
In baboons, thyroid hormone administration increases total beta-adrenergic receptor density by more than 100% (from 15.7±3.2 to 37.4±1.2 fmol/mg) and preferentially upregulates the beta-2 subtype (from 5.9±1.5 to 20.6±1.2 fmol/mg) over the beta-1 subtype (from 9.7±1.7 to 16.8±0.1 fmol/mg).
What the research says
1 studyStudy: Effects of thyroid hormone on cardiac beta-adrenergic responsiveness in conscious baboons.
Scientists gave baboons thyroid hormone and found that their heart receptors for adrenaline increased a lot—especially the type that responds to beta-2 signals. The numbers in the study match exactly what the claim says.
Score breakdown, mechanism chain, raw evidence, ideal studies needed & 1 supporting studies
Not medical advice. For informational purposes only. Always consult a qualified healthcare professional before making health decisions.