Visceral fat secretion of inflammatory cytokines induces insulin resistance, which promotes further visceral fat accumulation through inhibition of lipolysis and stimulation of de novo lipogenesis.

Study: Bilirubin reduces visceral obesity and insulin resistance by suppression of inflammatory cytokines

This study found that reducing inflammation in belly fat helps the body respond better to insulin, which in turn stops fat from building up. It shows that inflamed belly fat causes insulin problems, which makes more fat grow — just like the claim says.

Study: Greater fructose consumption is associated with cardiometabolic risk markers and visceral adiposity in adolescents.

This study found that eating lots of fructose (like in soda) leads to more belly fat, which then causes higher blood sugar and inflammation—exactly what the claim says happens when visceral fat gets too big. So even though the study didn’t directly test fat cells secreting chemicals, it shows the whole cycle in action.

Study: Apolipoprotein CIII Reduction Protects White Adipose Tissues against Obesity-Induced Inflammation and Insulin Resistance in Mice

This study found that reducing a specific protein in fat tissue lowers inflammation and helps the body respond better to insulin, which in turn lets fat cells break down fat normally again — supporting the idea that inflamed belly fat causes insulin problems that make more fat grow.

Study: Lipolysis in Health and Disease: Pathways, Regulation, and Metabolic Consequences

This study shows that when your body can't properly break down fat due to insulin resistance, fat builds up instead—exactly what the claim says. It’s like a broken faucet that won’t stop dripping fat into your organs.