The Claim

Elevated calcium concentrations of 10 mM induce a growth-phase-dependent activation of the glyoxylate shunt in Pseudomonas aeruginosa during stationary phase, resulting in increased levels of glyoxylate (3.47 LFC) and α-ketoglutarate (1.89 LFC), which reduces NADH production and conserves carbon.

Source: Calcium regulation of Pseudomonas aeruginosa metabolism.

What the research says

Supports is higher

Support is ahead, but a single strong opposing study can change this.

Supports
53score
Challenges
0score

These are independent scores, not a percentage. Higher-grade studies count more, so a single strong opposing study can outweigh several weaker ones.

How it works
1 study reviewed
In plain English

In Pseudomonas aeruginosa bacteria during stationary phase, high calcium levels trigger a metabolic shift that increases glyoxylate and α-ketoglutarate, reduces NADH production, and conserves carbon.

See the scientific wording

Elevated calcium concentrations (10 mM) induce a growth-phase-dependent activation of the glyoxylate shunt in Pseudomonas aeruginosa during stationary phase, increasing key metabolites such as glyoxylate (3.47 LFC) and α-ketoglutarate (1.89 LFC), which may enhance bacterial survival under oxidative stress by conserving carbon and reducing NADH production.

Why this might work

When calcium levels rise, the bacteria switch their energy production to a shortcut that saves carbon and makes less NADH, which helps them survive attacks from reactive oxygen molecules in the lungs.

Verified mechanismbased on 1 study

What the research says

1 study
  1. Study: Calcium regulation of Pseudomonas aeruginosa metabolism.

    When there’s a lot of calcium, like in the lungs of cystic fibrosis patients, this bacteria changes how it makes energy to save resources and better survive the stress — and the study proved it makes more of the key energy-saving molecule glyoxylate.

Score breakdown, mechanism chain, raw evidence, ideal studies needed & 1 supporting studies

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