The Claim

In human primary myoblasts derived from healthy young adults, treatment with 1 nmol/L of 1,25-dihydroxyvitamin D3 for 18 hours is associated with a 36% increase in vitamin D receptor (VDR) mRNA expression compared to untreated controls, suggesting a direct regulatory effect of active vitamin D on VDR gene expression in human muscle precursor cells.

Source: Effects of 1,25-Dihydroxyvitamin D3 and Vitamin D3 on the Expression of the Vitamin D Receptor in Human Skeletal Muscle Cells

What the research says

Supports is higher

Support is ahead, but a single strong opposing study can change this.

Supports
54score
Challenges
0score

These are independent scores, not a percentage. Higher-grade studies count more, so a single strong opposing study can outweigh several weaker ones.

How it works
1 study reviewed
In plain English

When scientists gave a specific form of vitamin D to muscle cells from young, healthy people for 18 hours, they noticed the cells made 36% more of a protein that helps vitamin D work — suggesting vitamin D might tell these cells to make more of this protein themselves.

See the scientific wording

In human primary myoblasts derived from healthy young adults, treatment with 1 nmol/L of 1,25-dihydroxyvitamin D3 for 18 hours is associated with a 36% increase in vitamin D receptor (VDR) mRNA expression compared to untreated controls, suggesting a direct regulatory effect of active vitamin D on VDR gene expression in human muscle precursor cells.

What the research says

1 study
  1. Study: Effects of 1,25-Dihydroxyvitamin D3 and Vitamin D3 on the Expression of the Vitamin D Receptor in Human Skeletal Muscle Cells

    Scientists gave human muscle precursor cells a small amount of active vitamin D for 18 hours and found the cells made 36% more of the vitamin D receptor protein, just like the claim said.

Score breakdown, mechanism chain, raw evidence, ideal studies needed & 1 supporting studies

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