The Claim

Higher parasympathetic nervous system activity, as measured by high-frequency heart rate variability (HF-HRV), is associated with per-cell changes in gene expression in myeloid immune cells—including classical and non-classical monocytes and neutrophils—that result in reduced expression of pro-inflammatory genes, independent of changes in the proportions of these immune cell types.

Source: Parasympathetic neural activity and the reciprocal regulation of innate antiviral and inflammatory genes in the human immune system

What the research says

Supports is higher

Support is ahead, but a single strong opposing study can change this.

Supports
44score
Challenges
0score

These are independent scores, not a percentage. Higher-grade studies count more, so a single strong opposing study can outweigh several weaker ones.

How it works
1 study reviewed
In plain English

When your body is more relaxed and your heart rate variability is high, your immune cells tend to turn down the volume on inflammation-related genes, even if the number of those cells doesn’t change.

See the scientific wording

Greater parasympathetic nervous system activity, indexed by high-frequency heart rate variability (HF-HRV), is associated with per-cell changes in gene expression in myeloid immune cells—particularly classical and non-classical monocytes and neutrophils—leading to reduced expression of pro-inflammatory genes, independent of shifts in immune cell proportions.

What the research says

1 study
  1. Study: Parasympathetic neural activity and the reciprocal regulation of innate antiviral and inflammatory genes in the human immune system

    When your body is calm and your heart rate variability is high, your nervous system sends signals to your immune cells to turn down inflammation genes and turn up virus-fighting genes—without changing how many of those cells you have.

Score breakdown, mechanism chain, raw evidence, ideal studies needed & 1 supporting studies

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