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Studies by this author
1 study attributed to Yuchen Bai on the platform. Click a card to read the full analysis.
Study
Abstract 4268: The allosteric MAT2A inhibitor IDE397 uniquely exploits metabolic liabilities associated with MTAP deletion to perturb DNA replication and repair
The study investigates how IDE397, an allosteric MAT2A inhibitor, exploits metabolic vulnerabilities in MTAP-deleted cancer cells by disrupting SAM-dependent pathways critical for DNA repair and PRMT5 inhibition.