The Claim

No patient with homozygous familial hypercholesterolemia (HoFH) achieved the European Atherosclerosis Society (EAS)-recommended low-density lipoprotein cholesterol (LDL-C) targets when treated with PCSK9 inhibitors in combination with standard lipid-lowering therapies, including statins, ezetimibe, and, in some cases, lipid apheresis.

Source: The Effect of PCSK9 Inhibitors on LDL-C Target Achievement in Patients with Homozygous Familial Hypercholesterolemia: A Retrospective Cohort Analysis

What the research says

Supports is higher

Support is ahead, but a single strong opposing study can change this.

Supports
33score
Challenges
0score

These are independent scores, not a percentage. Higher-grade studies count more, so a single strong opposing study can outweigh several weaker ones.

Description
1 study reviewed
In plain English

Even when people with a rare genetic cholesterol disorder take powerful new drugs called PCSK9 inhibitors along with their usual treatments, none of them got their bad cholesterol down to the recommended healthy levels.

See the scientific wording

No patient with homozygous familial hypercholesterolemia (HoFH) achieved European Atherosclerosis Society (EAS)-recommended LDL-C targets while receiving PCSK9 inhibitors in addition to standard lipid-lowering therapy, including statins, ezetimibe, and in some cases lipid apheresis.

What the research says

1 study
  1. Study: The Effect of PCSK9 Inhibitors on LDL-C Target Achievement in Patients with Homozygous Familial Hypercholesterolemia: A Retrospective Cohort Analysis

    The study looked at patients with a rare cholesterol condition who were taking PCSK9 inhibitors along with other cholesterol drugs, and none of them got their cholesterol low enough to meet the recommended targets.

Score breakdown, mechanism chain, raw evidence, ideal studies needed & 1 supporting studies

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