The Claim
Genetic tests that include the LCT-13910 polymorphism and other genetic variants can directly assess primary lactase persistence but cannot detect secondary lactose malabsorption caused by infectious or inflammatory bowel disease.
What the research says
Not yet evaluated
We are still looking at what the research says.
These are independent scores, not a percentage. Higher-grade studies count more, so a single strong opposing study can outweigh several weaker ones.
Genetic tests that analyze the LCT-13910 variant and other genetic markers can identify whether a person has inherited lactase persistence, but they cannot identify lactose malabsorption caused by infections or inflammatory bowel disease.
See the scientific wording
Genetic tests that include the LCT-13910 polymorphism and other genetic variants can directly assess primary lactase persistence, but they cannot detect secondary lactose malabsorption caused by infectious or inflammatory bowel disease.
People who inherit a specific genetic change keep making the enzyme that breaks down milk sugar throughout life, while those without it stop making it after infancy. This genetic trait cannot be changed by infections or gut inflammation, which only temporarily damage the gut lining and reduce enzyme levels without altering the genes.
What the research says
1 studyStudy: What is normal and abnormal in lactose digestion?
Genetic tests can tell if someone is naturally born able to digest milk, but they can't tell if someone developed milk intolerance because of a stomach infection or inflammation — those cases need different tests.
Score breakdown, mechanism chain, raw evidence, ideal studies needed & 1 supporting studies
Not medical advice. For informational purposes only. Always consult a qualified healthcare professional before making health decisions.