The Claim

The cytotoxic effect of cyclocreatine in C6 rat glioma cells is likely due to phosphocyclocreatine accumulation and subsequent cellular swelling, while its growth-inhibitory effects in OC238 human ovarian carcinoma cells occur through a different, unidentified mechanism that does not involve phosphocyclocreatine accumulation or cellular swelling.

Source: Cyclocreatine transport and cytotoxicity in rat glioma and human ovarian carcinoma cells: 31P-NMR spectroscopy.

What the research says

Roughly balanced

Support and challenge are close. The picture may shift as more studies come in.

Supports
6score
Challenges
0score

These are independent scores, not a percentage. Higher-grade studies count more, so a single strong opposing study can outweigh several weaker ones.

How it works
1 study reviewed
In plain English

Cyclocreatine kills brain tumor cells in rats by making them swell up, but it stops ovarian cancer cells in humans in a totally different way we don’t yet understand.

See the scientific wording

The cytotoxic effect of cyclocreatine in C6 rat glioma cells may result from phosphocyclocreatine accumulation and associated cellular swelling, whereas a different, unidentified mechanism must account for its growth-inhibitory effects in OC238 human ovarian carcinoma cells where neither phosphocyclocreatine accumulation nor swelling occurs.

What the research says

1 study
  1. Study: Cyclocreatine transport and cytotoxicity in rat glioma and human ovarian carcinoma cells: 31P-NMR spectroscopy.

    The study shows that cyclocreatine harms brain cancer cells by building up a toxic substance and making them swell, but it harms ovarian cancer cells in a different, unknown way.

Score breakdown, mechanism chain, raw evidence, ideal studies needed & 1 supporting studies

Fit Body Science verdict — we translate health claims into clear verdicts backed by peer-reviewed research.

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