Too much fat released from belly fat or made by the liver itself clogs liver cells, triggers inflammation, and makes the liver less responsive to insulin.

Chronic exposure of the liver to visceral adipose-derived free fatty acids and cytokines induces hepatic steatosis, elevates serum triglycerides, increases blood pressure, and promotes systemic insulin resistance.

When too many fat molecules and inflammatory signals from belly fat reach the liver, it becomes less responsive to insulin.

People with obesity have more fat circulating in their blood at rest, and their bodies are breaking down and re-storing fat at a faster rate than people without obesity.

Artificial trans fats make the liver produce more fat, cause more cell damage from oxidation, and trigger inflammation, which can lead to serious liver scarring over time.