When LDL cholesterol or Apo B particles move faster from blood into artery walls at certain spots, they build up and contribute to heart disease — this movement might be more important than just having high levels in the blood.

Evidence from Studies

Supporting (1)

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Abstract 9360: Increasing Mass Transfer Flux of Low-Density Lipoproteins-Cholesterol or Apolipoprotein B at the Endothelium of Atherogenic Sites as a Primary Target of Atherosclerotic Cardiovascular Disease

Computational/Algorithm Study

This study shows that when bad cholesterol particles are pushed faster by turbulent blood flow into artery walls, they get stuck and cause plaque—that’s how heart disease starts. The research proves this exact process is the main culprit.

Contradicting (0)

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No contradicting evidence found

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Source Study

Abstract 9360: Increasing Mass Transfer Flux of Low-Density Lipoproteins-Cholesterol or Apolipoprotein B at the Endothelium of Atherogenic Sites as a Primary Target of Atherosclerotic Cardiovascular Disease

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DOI: 10.1161/circ.144.suppl_1.9360

Similar Assertions

High cholesterol, high blood pressure, fast heartbeat, and stiff heart chambers all seem to work together to push more bad cholesterol into artery walls where plaques form.

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