NAC helps reduce stroke damage in some patients
Investigation of the effect of N-acetylcysteine on serum levels of oxidative inflammatory biomarkers in patients with stroke
Not medical advice. For informational purposes only. Always consult a healthcare professional. Terms
Surprising Findings
NAC only improved TOS in the overall group, but not other oxidative biomarkers
Many expected NAC to broadly improve multiple oxidative stress markers, but only TOS showed significant change in the full group.
Practical Takeaways
Stroke patients should discuss NAC with their doctor, but only as a potential supplement—not a replacement—for standard treatment.
Not medical advice. For informational purposes only. Always consult a healthcare professional. Terms
Surprising Findings
NAC only improved TOS in the overall group, but not other oxidative biomarkers
Many expected NAC to broadly improve multiple oxidative stress markers, but only TOS showed significant change in the full group.
Practical Takeaways
Stroke patients should discuss NAC with their doctor, but only as a potential supplement—not a replacement—for standard treatment.
Publication
Journal
Beni-Suef University Journal of Basic and Applied Sciences
Year
2023
Authors
Mohammad Farzandway, D. Elieh-Ali-Komi, Ehsan Mohammadi Noori, Farjam Goudarzi, Rezan Ashayeri Ahmadabad, A. Eshraghi, Z. Mirzaasgari, S. Navabi, A. Kiani
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Claims (10)
N-acetylcysteine (NAC) improves liver function, supports cellular detoxification, modulates inflammation in adipose tissue, enhances fat loss, and improves recovery from physical exertion.
N-acetylcysteine supplementation at 300-600 mg/day provides optimal benefits for most individuals, with diminishing returns at higher doses and potential gastrointestinal side effects.
N-acetylcysteine supplementation reduces exercise-induced oxidative stress in skeletal muscle by serving as a glutathione precursor, thereby improving recovery and maintaining performance during repeated exercise bouts.
N-acetylcysteine serves as a precursor to glutathione, the body's primary intracellular antioxidant and detoxification molecule.
Stroke patients who only received standard treatment showed no significant changes in blood markers related to oxidative stress over the first 24 hours of treatment.