Browse evidence-based analysis of health-related claims and assertions
For a few days, extra potassium lowers mice's blood pressure, but after three weeks, it starts to raise it again.
When mice eat a lot of potassium for three weeks, their blood pressure goes up mostly at night, no matter how much salt they eat or what form of potassium they consume.
Adults with type 2 diabetes who practiced yoga had about 0.5 lower BMI scores than those who didn't exercise, after 12 weeks.
Adults with type 2 diabetes who did aerobic exercise had about 0.6 lower BMI scores than those who didn't exercise, after 12 weeks.
Adults with type 2 diabetes who practiced yoga had about 17 less ng/mL of ICAM-1 (an inflammation marker) than those who didn't exercise, after 12 weeks.
Adults with type 2 diabetes who did aerobic exercise had about 27 less ng/mL of ICAM-1 (an inflammation marker) than those who didn't exercise, after 12 weeks.
Adults with type 2 diabetes who practiced yoga had about 0.6 lower insulin resistance scores than those who didn't exercise, after 12 weeks.
Adults with type 2 diabetes who did aerobic exercise had about 0.6 lower insulin resistance scores than those who didn't exercise, after 12 weeks.
Adults with type 2 diabetes who practiced yoga had about 13 more mg/dL lower fasting blood sugar than those who didn't exercise, after 12 weeks.
Adults with type 2 diabetes who did aerobic exercise had about 12 more mg/dL lower fasting blood sugar than those who didn't exercise, after 12 weeks.
Adults with type 2 diabetes who practiced yoga lost about 1.2 more kilograms than those who didn't exercise, after 12 weeks.
Adults with type 2 diabetes who did aerobic exercise lost about 1.5 more kilograms than those who didn't exercise, after 12 weeks.
In blood vessel cells under high sugar conditions, uric acid makes them produce more inflammation markers, but insulin doesn't have this effect.
When immune cells called monocytes are exposed to a bacterial toxin, adding uric acid and insulin together makes them produce more inflammation-related molecules than the toxin alone.
People with type 2 diabetes who take SGLT-2 inhibitors tend to have lower fasting insulin levels than those using other diabetes drugs.
Type 2 diabetes patients taking SGLT-2 inhibitors have lower levels of uric acid in their blood than those on other diabetes medications.
People with type 2 diabetes who take SGLT-2 inhibitors tend to have lower levels of a key inflammation marker called interleukin-6 than those taking other diabetes medications.
In Korean adults with healthy kidneys, the link between insulin resistance and kidney disease risk doesn't change based on age, gender, high blood pressure, or obesity status.
Korean adults with healthy kidneys who have higher leptin-to-adiponectin ratios (a marker of metabolic dysfunction) are 23% more likely to develop kidney disease over 11 years.
Korean adults with healthy kidneys who have lower QUICKI scores (indicating worse insulin sensitivity) have a 38% higher chance of developing kidney disease over 11 years compared to those with higher QUICKI scores.
For every point increase in HOMA-IR score (a measure of insulin resistance), Korean adults with healthy kidneys have a 13% higher chance of developing kidney disease over 11 years.
Korean adults with healthy kidneys who have high insulin resistance (HOMA-IR >1.9) are 40% more likely to develop kidney disease over 11 years compared to those with normal insulin resistance.
In kidney cells grown in a lab, insulin causes more URAT1 transporter to be present, which suggests insulin directly affects how kidneys handle uric acid. This finding is from the abstract summary - full study details were not available
In healthy rats, giving insulin makes the kidneys produce more URAT1 transporter and less ABCG2 transporter, which likely causes the kidneys to hold onto more uric acid instead of excreting it. This finding is from the abstract summary - full study details were not available