Health Claims

When teenage girls eat more processed foods, their breast tissue might look denser on scans only during the first half of their menstrual cycle, but this doesn't happen all the time or during other parts of the cycle.

PLIN2 protein levels in blood cells didn't show any clear relationship with common heart disease risk factors like cholesterol levels, blood sugar, or inflammation markers in people with heart disease.

In people having a heart attack, PLIN2 protein stays high even though the cellular cleanup system (proteasome) is more active, suggesting there might be special reasons why PLIN2 isn't being broken down properly during a heart attack.

In people with stable heart disease, older age is more strongly linked to higher PLIN2 protein levels than in people having a heart attack, suggesting age might affect PLIN2 differently in different types of heart disease.

In people with stable heart disease, when the proteasome (cellular cleanup system) is more active, there's less PLIN2 protein present, but this relationship doesn't hold for people having a heart attack, suggesting different ways PLIN2 is regulated in these two conditions.

The cellular cleanup system called the proteasome is more active in people having a heart attack compared to those with stable heart disease, which might be related to the body's response to the heart attack.

A protein called LAMP2A, which is involved in cellular cleanup processes, was found at similar levels in people having a heart attack and those with stable heart disease, and its levels didn't relate to PLIN2 protein levels.

The types and amounts of fats inside blood cells were similar between people having a heart attack and those with stable heart disease, even though the PLIN2 protein levels were different between these groups.

People having a heart attack have higher levels of harmful molecules called ROS/RNS in their blood compared to people with stable heart disease, which might be related to the heart attack process.

In people with heart disease, older patients tend to have higher levels of PLIN2 protein in their blood cells, and this relationship is stronger in those with stable heart disease than in those having a heart attack.

In people with stable heart disease, when the proteasome (a cellular cleanup system) is more active, there's less PLIN2 protein present, but this relationship doesn't hold for people having a heart attack.

In people having a heart attack called STEMI, the amount of a protein called PLIN2 in certain blood cells is higher than in people with stable heart disease, which might be related to why their heart attack happened.

Mice that got the supplement were more responsive to insulin compared to mice that didn't get it.

Mice that got the supplement had less fat buildup in their brown fat compared to mice that didn't get it.

Mice that got the supplement had higher levels of genes that help burn fat in their livers compared to mice that didn't get it.

Mice that got the supplement had much lower levels of genes involved in fat uptake and storage in their livers compared to mice that didn't get it.

Mice that got the supplement had higher levels of genes that help burn calories in their brown fat compared to mice that didn't get it.

Mice that got the supplement had lower levels of a gene that helps store fat in their livers compared to mice that didn't get it.

Mice that got the supplement had much lower levels of fat and cholesterol in their livers compared to mice that didn't get it.

Mice that got the supplement had smaller livers compared to mice that didn't get it.

Mice that got the supplement had much lower levels of insulin in their blood compared to mice that didn't get it.

Mice that got the supplement were better able to handle sugar in their blood after eating.

Mice that got the supplement had higher levels of a hormone that helps regulate metabolism in their fat tissue.

Mice that got the supplement had fewer immune cells in their fat tissue compared to mice that didn't get it.