Browse evidence-based analysis of health-related claims and assertions
In mice, a drug called fenofibrate helps protect the brain after anesthesia, but only if the brain can burn fat for energy — when another drug blocks fat burning, the protection goes away.
Giving mice a drug called fenofibrate before anesthesia might boost brain proteins that help burn fat for energy — like turning up the brain’s fuel-burning system for several days after the procedure.
Giving mice a specific drug before anesthesia helps them remember scary situations better after waking up.
In male mice, a long dose of a common anesthesia gas seems to lower levels of brain proteins that help burn fat for energy, and this effect lasts up to a week after the anesthesia wears off.
Mice given a common anesthesia for 6 hours remember scary situations worse for at least a week after, showing they’re less fearful when they should be — a sign their memory is temporarily impaired.
Scientists are looking into whether changing the bacteria in your gut—using things like probiotics, prebiotics, or even poop transplants—could help treat obesity and diabetes.
The bacteria in your gut might affect your weight and blood sugar by changing how your body uses energy and handles inflammation.
The mix of bacteria in your gut might be connected to conditions like obesity and diabetes, and certain types of bacteria could be tied to storing more fat and how your body handles sugar.
Fenofibrate, a drug that changes how the liver processes energy, seems to help reduce liver cysts and damage in a type of mouse with a genetic liver disease.
In mice with a genetic form of kidney disease, taking a drug called fenofibrate seems to slow down the growth of kidney cysts and reduce harmful inflammation in the kidneys.
Fenofibrate seems to slow down kidney disease in mice with a condition similar to polycystic kidney disease, making their kidneys healthier after five months of treatment.
In mice with a kidney disease similar to one in humans, giving them a drug called fenofibrate seems to boost the activity of genes that help burn fat for energy in the kidneys, which might mean their kidney cells are using energy more efficiently.
In liver cells from rats, blocking a key energy sensor called AMPK stops acetic acid from changing fat levels and fat storage — suggesting that acetic acid needs this sensor to work.
Acetic acid helps reduce fat buildup in rat liver cells, and this only happens when a specific energy-sensing pathway (AMPK) is working — if you block that pathway, the effect goes away.
Acetic acid might slow down fat-making in rat liver cells by turning off key fat-related genes.
Treating rat liver cells with low doses of acetic acid boosts genes and proteins that help burn fat, suggesting these cells might get better at breaking down fats.
When rat liver cells are treated with a specific amount of sodium acetate, it turns on a key energy-sensing switch in the cells within a few hours.
In cow liver cells, a chemical called acetate seems to help reduce fat buildup, especially when more is used, and this effect goes away if a specific cellular switch (AMPK) is turned off.
In cow liver cells, a chemical called acetate seems to turn off a key fat-making enzyme by flipping a molecular switch, and this only happens when another switch (called AMPK) is working.
When cow liver cells are exposed to certain levels of a substance called acetate, it seems to turn down the activity of genes that tell the cell to make fat — and this might happen through a specific cellular switch called AMPK.
In cow liver cells, a substance called acetate boosts a protein that helps burn fat, and the more acetate you add, the stronger the effect — but this only happens when another key energy-sensing switch is turned on.
When cow liver cells are exposed to certain levels of a substance called sodium acetate, it turns on a key energy sensor in the cells, and this effect gets stronger over 3 hours and goes away if you block that sensor.
Giving a specific amount of vinegar-like acid to cow liver cells in a lab seems to cut fat buildup by a third — this might mean it helps prevent fatty liver in cows.
In cow liver cells, a certain amount of a chemical called sodium acetate seems to turn off a key fat-making enzyme, and this might be controlled by another switch called AMPK.