Browse evidence-based analysis of health-related claims and assertions
The fat cells in mice that got the supplement were about 30% smaller than in mice that didn't get it.
Mice that got the supplement had smaller fat deposits around their organs and under their skin compared to mice that didn't get it.
Mice that got the supplement had about half as much body fat as mice that didn't get it, even when both groups ate the same high-fat diet.
Giving mice a special supplement in their water for 11 weeks helped them stay lighter when eating a high-fat diet, even though they ate the same amount of food.
For some stroke patients, NAC treatment actually made some blood markers related to oxidative stress worse, while improving others.
When comparing stroke patients who received NAC to those who didn't, only one blood marker related to oxidative stress showed a significant difference between the groups.
The study did not measure whether NAC improves thinking skills or memory in stroke patients, as it only measured stroke severity symptoms.
Stroke patients who only received standard treatment showed no significant changes in blood markers related to oxidative stress over the first 24 hours of treatment.
For most stroke patients, NAC treatment didn't significantly change most blood markers related to oxidative stress compared to standard treatment alone.
For some stroke patients, NAC treatment significantly improves several blood markers related to oxidative stress and inflammation.
Stroke patients who receive NAC along with standard treatment show better recovery from stroke symptoms over time compared to those who only get standard treatment.
Taking NAC along with regular stroke treatment lowers a measure of harmful oxidative stress in the blood of stroke patients.
Adequate protein intake preserves muscle mass, which maintains metabolic rate.
Specific intermittent fasting patterns (one 36-hour fast per week, two non-consecutive 24-hour fasts, or three separate 16-hour fasts) increase norepinephrine, fat oxidation, and glucose tolerance while avoiding metabolic downshift.
Intermittent fasting without daily fasting maintains higher metabolic rate while still providing metabolic benefits.
Daily 16:8 fasting can lead to a metabolic slowdown within 2-3 weeks due to unintentional caloric restriction.
Non-consecutive 24-hour fasting three times per week improves glucose tolerance.
Prolonged fasting (72 hours) impairs glucose tolerance, resulting in higher blood glucose and insulin responses to carbohydrate intake.
Caloric restriction for 3 weeks results in a 266-calorie per day reduction in resting metabolic rate, with approximately half of this reduction due to adaptive thermogenesis.
Frequent or prolonged fasting triggers adaptive thermogenesis, causing the body to reduce metabolic rate to conserve energy.
Metabolic rate increases during 24-36 hour fasts due to norepinephrine and adrenaline release, but does not continue to increase beyond 36 hours of fasting.
Prolonged or frequent fasting can cause a decrease in metabolic rate after an initial increase.
Because PGC-1alpha helps control fat metabolism, it might be useful for developing treatments for obesity and type 2 diabetes
PGC-1alpha helps control how cells use energy